neurotoxin (Also neurotoxins) : Related Words Words similar in meaning to neurotoxin

  • neurolysin«
  • cholinesterase inhibiting substance«
  • bacterial neurotoxin«
  • toxic substance«
  • diseases«
  • neurotoxin«
  • tetrodotoxin«
  • botulinum toxin«
  • domoic acid«
  • saxitoxin«
  • kokoi venom«
  • botulinum toxin A«
  • tremortin«
  • neuron«
  • neurotoxic«
  • toxin«
  • brain«
  • synapses«
  • anatoxin«
  • scyllatoxin«
  • nervous system«
  • sarin«
  • pufferfish«
  • compound«
  • penitrem«
  • nachrs«
  • oxidopamine«
  • neuron communication«
  • neurotoxigenic«
  • α-bungarotoxin«
  • neurotoxicology«
  • neurotoxicant«
  • neurotoxicity«
  • neurotoxically«
  • inhibition«
  • mechanism«
  • neurotoxic effect«
  • bungarotoxin«
  • methylazoxymethanol acetate«
  • glutamate«
  • methiocarb«
  • neuroscience«
  • maurotoxin«
  • receptor«
  • mandaratoxin«
  • sodium channel«
  • latrotoxin«
  • apoptosis«
  • imiprothrin«
  • hefutoxin«
  • function«
  • gabaculine«
  • ethanol«
  • fugu«
  • neurotoxin exposure«
  • clinical neurotoxicology«
  • dimethylmercury«
  • nitric oxide«
  • dendrotoxin«
  • lead«
  • coniine«
  • toxicity«
  • charybdotoxin«
  • ability«
  • cangitoxin«
  • calcium«
  • blowfish«
  • apamin«
  • caramboxin«
  • effect«
  • neuromuscular junction«
  • tent«
  • vivo«
  • calcium flux«
  • conotoxins«
  • endogenous compound«
  • toxic effect«
  • ion channel«
  • hippocampus«
  • puffer fish«
  • postsynaptic neuron«
  • poison«
  • ingestion«
  • arsenic«
  • nicotinic acetylcholine receptor«
  • nervous tissue«
  • mitochondrial transition«
  • chronic ethanol intake«
  • tea«
  • paralysis«
  • astrocyte«
  • nmda receptor«
  • ttx exposure«
  • iadecola«
  • potassium channel«
  • disease«
  • tetanus«
  • endogenous neurotoxin«
  • mehg«
  • fast death factor«
  • chlorotoxin«
  • antioxidant level«
  • toxicological review«
  • diethylmercury«
  • ammonia toxicity«
  • action potential«
  • administration«
  • glutamate excitotoxicity«
  • antioxidant«
  • resultant death«
  • dopamine«
  • tetraethylammonium«
  • united state environmental protection agency«
  • blood flow«
  • star fruit«
  • inhibitory neuron«
  • blood«
  • spinal cord«
  • muscular atrophy«
  • arsenite«
  • fetal brain«
  • poisoning«
  • fetus«
  • ammonia«
  • result«
  • potent neurotoxin«
  • cytotoxin«
  • choroid plexus«
  • nerve tissue«
  • usepa«
  • cerebral edema«
  • fetal alcohol syndrome«
  • curare«
  • blood brain barrier«
  • nmda«
  • muscular contraction«
  • organophosphate«
  • defect«
  • cell«
  • neurogenesis«
  • response«
  • dependent manner«
  • disruption«
  • ncbi«
  • cytoskeleton«
  • muscle«
  • damaging effect«
  • musculature«
  • muscle cell«
  • acetylcholine«
  • ach«
  • oxidative stress«
  • capillary«
  • organism«
  • no«
  • body«
  • symptom«
  • necrosis«
  • neurotransmitter«
  • survival«
  • concentration«
  • exposure«
  • form«
  • susceptibility«
  • inter«
  • vitro«
  • significant improvement«
  • epilepsy«
  • significant damage«
  • κ-bungarotoxin«
  • α7-nachr.«
  • α7-nachr function«
  • widespread nerve function interruption«
  • widespread central nervous system damage«
  • widespread brain atrophy«
  • vitro neurotoxicology«
  • vesicular transport inhibition«
  • vesicular neurotransmitter«
  • ttx consumption«
  • true neurotoxin«
  • toxin exposure symptom«
  • toxin absorption«
  • tight hydrophobic layer«
  • though nitric oxide«
  • tent migration«
  • tea function«
  • systemic nervous system arrest«
  • systemic muscular contraction«
  • systemic aluminum level«
  • susceptible subset«
  • susceptible nature«
  • subsequent neuronal death«
  • spencer p«
  • single generic sodium«
  • significant neuron death«
  • significant lead exposure«
  • significant functional disruption«
  • selective neurotoxins«
  • selective neurotoxin«
  • schaumburg hh«
  • s. r. karim«
  • ronald d. poretz«
  • robert b. tallitsch«
  • resultant calcium ion flow«
  • reduced motor coordination«
  • reduced excitability«
  • proper excitability«
  • previous ethanol experience«
  • potential invasive harm«
  • potential chemical insult«
  • potassium channel responsible«
  • potassium channel activity«
  • permanent parkinsonism«
  • paralytic complication«
  • oxford medical journal«
  • ornella rossetto«
  • original hodgkin«
  • nutrient entry«
  • nonpeptide amino acid toxin«
  • nociception neuron«
  • nitric oxide/superoxide«
  • neurotoxin treatment«
  • neurotoxin testing«
  • neurotoxin institute«
  • neurotoxin category«
  • neurotoxicity result«
  • neurotoxic form«
  • neurotoxic causative compound«
  • neurotoxic behavior«
  • neuronal membranes?"trends«
  • neuronal cytotoxicity«
  • neuron response contribution«
  • neuron excitotoxicity«
  • neuron control«
  • neuron cellular process«
  • neurofilament transport«
  • neurofilament motility«
  • nervous tissue death«
  • nervous cell remodeling«
  • muscular tetany«
  • muscarinic acetylcholine antagonist atropine«
  • multiple effects«
  • mitochondrial transmembrane potential«
  • mitchell f«
  • michael j. timmons«
  • metcalf j«
  • membrane depolarization mechanism«
  • mediated(pkg«
  • mature nervous tissue«
  • marina guizzetti«
  • manganese glutamate«
  • m. a. baker«
  • ludolph ac«
  • lucio g.«
  • ltp increase«
  • low ph induced entry«
  • lithium administion«
  • lethal volume«
  • leslie r. whitaker«
  • krait snake«
  • kevin t. strang«
  • john e. maggio«
  • ischemic region«
  • ion channel disruption«
  • intracellular calcium ion accumulation«
  • intense insult«
  • intact inter«
  • insufficient tidal volume«
  • inhibited process«
  • ingested bungarotoxin«
  • induced delayed polyneuropathy«
  • hitoshi morikawa«
  • hexane«
  • hershel raff«
  • healthy nervous tissue«
  • health effect test guideline«
  • haussinger«
  • h a m n«
  • glutamine activity«
  • glutaminase inhibitor«
  • glial cell damage«
  • gary r. strichartz«
  • g.b. van der voet«
  • f.a. de wolff«
  • extensive plumbing network«
  • extensive inroad«
  • exogenous neurotoxin«
  • exogenous chemical neurological insult«
  • ethanol neurotoxicity«
  • ethanol exposure result«
  • environmental neurotoxin arsenic impairs neurofilament dynamic«
  • endogenous neurotoxin sources«
  • endogenous ailment«
  • effective toxin exposure«
  • e. a. gazi«
  • double anchorage«
  • distinct poison«
  • distinct event induced«
  • diminished neural network function«
  • dietary vitamin e result«
  • destructive compound«
  • deleterious compound«
  • david w. eaves«
  • cytotoxicity function«
  • cytoskeletal modification«
  • craniofacial formation«
  • cortical cell culture«
  • continuous ethanol consumption«
  • conotoxin«
  • common morphogenesis aberration«
  • codd ga«
  • cltx result«
  • clifford c. shone«
  • chronic tea administration«
  • chronic ethanol ingestion«
  • chloride channel ligand«
  • chloride channel«
  • chinese electronics factory«
  • chain alpha form«
  • cellular membrane constituent«
  • cell mechanism interference«
  • calcium channel«
  • c. s. bala«
  • btx function«
  • btx antitoxin«
  • botulinum toxin bind«
  • bonfoco«
  • bbb. mercury«
  • basic cell structure«
  • astrocyte remodling«
  • astrocyte remodeling«
  • arthur j. vander«
  • arsenic neurotoxicity a review«
  • antitoxin administration«
  • angelo moretto«
  • ammonia activity«
  • aluminium«
  • aleksander r. smith«
  • alcohol inhibition«
  • ahasan«
  • adverse compound«
  • adolescent alcohol abuse model«
  • ach vesicle«
  • acetylcholinesterase inhibitor neostigmine«
  • acetylcholinesterase capacity«
  • a. a. mamun«
  • central nervous system«
  • sodium«
  • venom«
  • vitro testing environment«
  • venomous organism«
  • vahidnia«
  • tetradontidae«
  • tetanus neurotoxin«
  • systemic metabolism«
  • subsequent suffocation«
  • spasticity disorder«
  • selective passage«
  • potent excitatory«
  • potassium flux«
  • persistent memory impairment«
  • paolo bolognese«
  • nmda activation«
  • neurotoxicology research«
  • neurite growth inhibition«
  • nervous tissue function«
  • mercury«
  • lead neurotoxicity«
  • jun terminal kinase«
  • intracellular calcium ion level«
  • hippocampal neurodegeneration«
  • gennaro giordano«
  • extensive usefulness«
  • defuria«
  • cytotoxicity«
  • concentrated lead«
  • clostridial neurotoxin«
  • cardiovascular formation«
  • arsenic ingestion«
  • ampa glutamatergic ionotropic receptor«
  • active compound responsible«
  • system«
  • wenbin«
  • ventral tegmental area«
  • venom capable«
  • stephanie a.«
  • sodium channel function«
  • singapore medical journal«
  • severe developmental defect«
  • severe deleterious effect«
  • peripheral nervous system damage«
  • neuron action potential«
  • neurodegenerative property«
  • neural migration«
  • muscle cell contract«
  • mesolimbic dopamine neuron«
  • mercuric ion«
  • membrane concentration«
  • marine cone snail«
  • local pathology«
  • lidsky«
  • intracellular calcium increase«
  • inhibitory transmission«
  • impaired learning«
  • excitotoxic effect«
  • endogenous glutamate«
  • diameter axon«
  • developmental neurotoxicity«
  • curare poison«
  • breathing stop«
  • bicyclic amine alkaloid«
  • astrocyte function«
  • adverse neurological effect«
  • contraction«
  • support«
  • aluminum«
  • membrane«
  • ω-conotoxin«
  • theodore i.«
  • reward expectation«
  • protective cell«
  • patient incapable«
  • neurotoxic compound«
  • neuron synapsis«
  • natural neurotransmitter«
  • lead pan«
  • fragile cell«
  • environmental neurotoxin«
  • direct lethality«
  • chain disulfide bond«
  • cesare montecucco«
  • brain energy metabolism«
  • nutrient«
  • voltage«
  • vivo capability«
  • toxipedia«
  • sympathetic nerve activity«
  • specific categorization«
  • skeletal modification«
  • pearson/benjamin cummings«
  • inositol 1,4,5-triphosphate«
  • depolarization phase«
  • cyanobacterial toxin«
  • accurate distinction«
  • characterization«
  • usepa.«
  • reactive oxidative specie«
  • nmda receptor activity«
  • neurotransmitter transport«
  • michael r«
  • memory acquisition«
  • lipophilic toxin«
  • kimberly nixon«
  • external assault«
  • ethanol exposure«
  • a. trevor«
  • target channel«
  • receptor channel«
  • montecucco«
  • macroscopic manifestation«
  • intracellular function«
  • experimental toxicology«
  • distinct compound«
  • liver«
  • uniform environment«
  • subsequent cell death«
  • pirazzini«
  • oligodendroglia«
  • ischemic brain injury«
  • hydrophilic compound«
  • huxley model«
  • debin«
  • significant inhibition«
  • environmental genomics«
  • day understanding«
  • calcium ion influx«
  • bungarotoxins«
  • bacterium clostridium botulinum«
  • reorganization«
  • neural component«
  • extensive class«
  • excess ion«
  • clinical correlate«
  • calcium atpase«
  • mature organism«
  • aluminum toxicity«
  • development«
  • functionality«
  • progressive symptom«
  • muscular relaxation«
  • predator«
  • cns damage«
  • chronic ingestion«
  • botulinum neurotoxin«
  • protein kinase g«
  • porcupine fish«
  • neuron death«
  • dependent potassium channel«
  • cellular microbiology«
  • vivo system«
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