estradiol (Also estradiols) : Related Words Words similar in meaning to estradiol
- oestradiol«
- phenol«
- estrogen«
- chemical compound«
- estradiol«
- vertebrate«
- trioxifene«
- e2«
- oestrogen«
- synthetic«
- estrone«
- ovary«
- orestrate«
- estriol«
- hormone«
- comt«
- gonadarche«
- er«
- follicular phase«
- fluorestradiol«
- ethinylestradiol«
- estrogenic«
- δ4-androstenedione«
- cyp1b1«
- estratetraenol«
- estrogen receptor«
- estrapronicate«
- pregnancy«
- cloxestradiol«
- breast cancer«
- luteal phase«
- liver«
- inactivation«
- menstrual cycle«
- gper«
- cyp2c8«
- estradiol level«
- menopause«
- methylation«
- induction«
- erα«
- cyp1a2«
- cyp2c9«
- aromatase«
- dehydrogenation«
- dna«
- serum level«
- cyp1b1 expression«
- adenohypophyseal hormone secretion«
- cyp3a4«
- inhibition«
- quinone«
- estradiol metabolite«
- estetrol«
- aromatase activity«
- testosterone«
- metabolite«
- cyp1a1«
- steroid«
- erβ«
- estrogenic activity«
- sperm count«
- brain«
- estrogen level«
- superoxide«
- nuclear receptor«
- hypothalamic«
- cytochrome p450«
- woman«
- endometrium«
- gene transcription«
- breast«
- tissue«
- ovulation«
- angiogenesis«
- androgen«
- progesterone«
- level«
- enzyme«
- e1«
- hydroxyl«
- agonist«
- cholesterol«
- uterus«
- evidence«
- lining«
- bone«
- apoptosis«
- dna.«
- vagina«
- puberty«
- modulation«
- conversion«
- δ4- pathway«
- woman past menopause experience«
- weak carcinogenicity«
- weak carcinogenic activity«
- vascular smooth muscle growth«
- theca folliculi cell«
- steroid carbonyl«
- slow depurination half«
- situ estradiol biosynthesis«
- similar redox potential«
- similar redox cycling activity«
- sex chromosome genetic condition«
- several benign gynecologic condition«
- relative estrogen deficiency«
- reductase catalysis«
- reactive hydroxyl radical capable«
- ranges«
- protein exhibit constitutive expression«
- prostaglandin pge2«
- potent er agonist«
- placental production«
- pituitary event«
- ovarian estradiol«
- other hydroxylations«
- myometrial oxytocin receptor«
- myometrial growth«
- microsomal cyps«
- menstrual level«
- menstrual female reproductive cycle«
- mammary fibroblast«
- male reproduction«
- major hepatic pathway«
- lipoidal estradiol form«
- leiomyomata uterus«
- individual laboratory result«
- h¬-ras«
- hypoestrogenicity«
- hormone surge«
- hamster kidney tumor model«
- gynecological cancers«
- glucuronide formation«
- genotoxic quinone formation«
- further antitumor activity«
- follicle trigger«
- female reproduction«
- extrahepatic tissue shift«
- extrahepatic 2-hydroxylation«
- estrogen redox cycling«
- estrogen monitoring«
- estrogen homeostasis«
- estrogen classical pathway«
- estrane steroid ring system«
- estradiol-3,4-semiquinone«
- estradiol-3,4-quinone«
- estradiol metabolism«
- estradiol level measure«
- estradiol increase due«
- estradiol 4-hydroxylation«
- er activation«
- electrophilic dna.«
- e2 methylation«
- e2 metabolite«
- e2 inactivation«
- e2 formation«
- e2 classical pathway«
- dependent carcinogenesis«
- cytokine receptor synthesis«
- cyp1b1 mrna«
- cyp1b1 ere«
- common extrahepatic pathway«
- cellular proliferation stimulation«
- bone mass due«
- baseline estrogen«
- base excision repair mechanism«
- basal aromatase activity«
- approximate typical female brain structure«
- adult male sexual behavior«
- additional mers«
- active precursor«
- accelerated loss«
- -diol«
- soluble conjugate«
- semiquinone intermediate«
- quinone formation«
- primary male sex hormone«
- pituitary system«
- pathway independent«
- nuclear steroid hormone receptor«
- neuroprotective function«
- menstrual dysfunction«
- immature mammal«
- genetic upregulation«
- fertility therapy«
- female reproductive tissue«
- fat structure«
- estradiol support«
- estradiol molecule«
- estradiol act«
- dimorphic brain structure«
- dihydroxyestrin«
- depression postpartum«
- cyp3a isoforms«
- cervical softening«
- absolute serum level«
- oxidation«
- upregulating«
- unstable adduct«
- skin composition«
- sexual development«
- quinone reductase«
- predominant estrogen«
- p450 reductase«
- metabolic scheme«
- measurement«
- increased reactivity«
- estrogen receptor β«
- estrogen exposure«
- cytokine il-6«
- correct regulation«
- carcinogenic capacity«
- antimetastatic effect«
- blockade«
- unintentional ingestion«
- prone repair«
- male sperm cell«
- major urinary metabolite«
- g mutation«
- estrogen receptor α«
- cervical gland«
- transferase activity«
- significant mood«
- mammalian testis«
- glycosyl bond«
- fetal adrenal gland«
- estradiol stearate«
- estradiol palmitate«
- kidney«
- steroid precursor«
- serum estradiol«
- primary female sex hormone«
- potent nucleophile«
- paracrine action«
- metabolic transformation«
- intracellular signalling«
- estrogen response element«
- estradiol production«
- tumor blood supply«
- situ synthesis«
- clinical recovery«
- active estrogen«
- uterine blood flow«
- effect independent«
- breast cancer tumor«
- significant role«
- schiff base formation«
- positive feedback system«
- membrane estrogen receptor«
- key intermediary«
- genomic effect«
- follicular growth«
- erx«
- additional pathway«
- fat«
- semiquinone«
- redox cycling«
- glutathione conjugate«
- comt inhibitor«
- apurinic site«
- function«
- bean«
- tumorigenic«
- prolonged activation«
- principal pathway«
- postmenopause«
- fetal health«
- connection«
- prostaglandin production«
- metabolic fate«
- mer.«
- follicular development«
- female ovary«
- chain cleavage«
- strand dna break«
- endogenous estrogen«
- arterial blood flow«
- epiphyseal closure«
- dissociation rate«
- radical formation«
- endocrine tissue«
- androgen deprivation«
- normal menstrual cycle«
- c3 position«
- other effects«
- estrane«
- predominant mechanism«
- breast growth«
- decrease«
- prenatal life«
- fat deposition«
- caspase-8«
- minor pathway«
- consumption«
- uterine bleeding«
- superoxide radical«
- cellular transformation«
- antiestrogen«
- prohormones«
- human breast cancer cell«
- responsive gene«
- cancer cell proliferation«
- sex differentiation«
- physiological consequence«
- perimenopause«
- semen analysis«
- carcinogenic potential«
- covalent linkage«
- estrogen production«
- lab test online«
- smaller amount«
- cancer tissue«
- coadministration«
- pregnancy loss«
- caspase-3«
- form structure«
- nitrogenous base«
- estrogenic effect«
- immunomodulation«
- enterohepatic circulation«
- klinefelter«
- dna fragmentation«
- biological activity«
- a ring«
- shock protein«
- autocrine«
- antitumor activity«
- sequential step«
- myometrium«
- aromatization«
- multiple protein«
- human sexual behavior«
- cellular growth«
- diethylstilbestrol«
- theelin«
- stilboestrol«
- stilbestrol«
- oestrone«
- oestriol«
- mestranol«
- hexestrol«
- diethylstilboestrol«
- 2-meoe2«
- 4-ohe2«
- 16α-ohe2«
- 2-ohe2«
- 1-n7gua«
- 4-androstenedione«
- 17β-hydroxysteroid dehydrogenase«
- 4-meoe2«
- 3,4-q level«
- 16α-ohe1«
- 1-n3ade exhibit instantaneous depurination«
- 2-hydroxyestradiol«
- 17α-estradiol«
- Estronol«
- DES«
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